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Chylomicrons alter the fate of endotoxin, decreasing tumor necrosis factor release and preventing death.

The hypertriglyceridemia of infection was traditionally thought to represent the mobilization of substrate to fuel the body's response to the infectious challenge. However, we have previously shown that triglyceride-rich lipoproteins can protect against endotoxin-induced lethality. The current...

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Autores principales: Harris, H W, Grunfeld, C, Feingold, K R, Read, T E, Kane, J P, Jones, A L, Eichbaum, E B, Bland, G F, Rapp, J H
Formato: Artigo
Lenguaje:Inglês
Publicado: 1993
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC288056/
https://ncbi.nlm.nih.gov/pubmed/8450032
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