Interplay between REST and nucleolin transcription factors: a key mechanism in the overexpression of genes upon increased phosphorylation

Non-malignant cells can be transformed via the activation of kinases that control degradation of neural-restrictive silencer factor (REST). Here, we identify a mechanism that contributes to the activation of genes, expression of which is controlled by responsive elements containing overlapping bindi...

תיאור מלא

שמור ב:
מידע ביבליוגרפי
Main Authors: Tediose, Teeo, Kolev, Martin, Sivasankar, Baalasubramanian, Brennan, Paul, Morgan, B. Paul, Donev, Rossen
פורמט: Artigo
שפה:Inglês
יצא לאור: Oxford University Press 2010
נושאים:
Gene Regulation, Chromatin and Epigenetics
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC2875004/
https://ncbi.nlm.nih.gov/pubmed/20100803
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/nar/gkq013
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