Interleukin-1β Increases Gap Junctional Communication among Synovial Fibroblasts via the Extracellular Signal Regulated Kinase Pathway

BACKGROUND INFORMATION: The gap junction protein, connexin43, has been implicated in the etiology of osteoarthritis. Work from others has revealed that the size and number of gap junctions increases in synovial biopsies from patients with osteoarthritis. Further, pharmacologic inhibition of connexin...

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Hlavní autoři: Niger, Corinne, Howell, Floyd D., Stains, Joseph P.
Médium: Artigo
Jazyk:Inglês
Vydáno: 2009
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Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC2874634/
https://ncbi.nlm.nih.gov/pubmed/19656083
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1042/BC20090056
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