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Chronic administration of atypical antipsychotics improves behavioral and synaptic defects of STOP null mice
Recent studies have suggested that schizophrenia is associated with alterations in the synaptic connectivity involving cytoskeletal proteins. The microtubule-associated protein STOP (Stable Tubule Only Polypeptide) plays a key-role in neuronal architecture and synaptic plasticity and it has been dem...
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| 主要な著者: | , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Springer-Verlag
2010
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2874572/ https://ncbi.nlm.nih.gov/pubmed/19936716 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00213-009-1712-3 |
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