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Modulation of chronic hypoxia-induced chemoreceptor hypersensitivity by NADPH oxidase subunits in rat carotid body

Previous studies in our laboratory established that reactive oxygen species (ROS) generated by NADPH oxidase (NOX) facilitate the open state of a subset of K(+) channels in oxygen-sensitive type I cells of the carotid body. Thus pharmacological inhibition of NOX or deletion of a NOX gene resulted in...

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Bibliographische Detailangaben
Hauptverfasser: He, L., Liu, X., Chen, J., Dinger, B., Stensaas, L., Fidone, S.
Format: Artigo
Sprache:Inglês
Veröffentlicht: American Physiological Society 2010
Schlagworte:
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2867529/
https://ncbi.nlm.nih.gov/pubmed/20185631
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/japplphysiol.00766.2009
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