KChIP2 Attenuates Cardiac Hypertrophy Through Regulation of I(to) and Intracellular Calcium Signaling

Recent evidence shows that the auxiliary subunit KChIP2, which assembles with pore-forming Kv4-subunits, represents a new potential regulator of the cardiac calcium-independent transient outward potassium current (I(to)) density. In hypertrophy and heart failure, KChIP2 expression has been found to...

Full description

Saved in:
Bibliographic Details
Main Authors: Jin, Hongwei, Hadri, Lahouaria, Palomeque, Julieta, Morel, Charlotte, Karakikes, Ioannis, Kaprielian, Roger, Hajjar, Roger, Lebeche, Djamel
Format: Artigo
Language:Inglês
Published: 2010
Subjects:
Article
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC2866822/
https://ncbi.nlm.nih.gov/pubmed/20051248
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2009.12.019
Tags: Add Tag
No Tags, Be the first to tag this record!

Similar Items