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KChIP2 Attenuates Cardiac Hypertrophy Through Regulation of I(to) and Intracellular Calcium Signaling
Recent evidence shows that the auxiliary subunit KChIP2, which assembles with pore-forming Kv4-subunits, represents a new potential regulator of the cardiac calcium-independent transient outward potassium current (I(to)) density. In hypertrophy and heart failure, KChIP2 expression has been found to...
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| Autores principales: | , , , , , , , |
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| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
2010
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2866822/ https://ncbi.nlm.nih.gov/pubmed/20051248 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2009.12.019 |
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