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Overexpression of amyloid precursor protein A4 (beta-amyloid) immunoreactivity in genetically transformed cells: implications for a cellular model of Alzheimer amyloidosis.

Among the major obstacles to clarifying molecular mechanisms involved in amyloid metabolism in Alzheimer disease has been the unavailability of laboratory models for this uniquely human disorder. The present studies were aimed at establishing genetically engineered cell lines that overexpress amyloi...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Egile Nagusiak: Marotta, C A, Chou, W G, Majocha, R E, Watkins, R, LaBonne, C, Zain, S B
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: 1989
Gaiak:
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC286459/
https://ncbi.nlm.nih.gov/pubmed/2563163
Etiketak: Etiketa erantsi
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