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Adenosine A(2A) Receptor Activation and Macrophage-mediated Experimental Glomerulonephritis
In immune-induced inflammation, leukocytes are key mediators of tissue damage. Since A(2A) adenosine receptors (A(2A)R) are endogenous suppressors of inflammation, we examined cellular and molecular mechanisms of kidney damage to determine whether selective activation of A(2A)R will suppress inflamm...
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| Auteurs principaux: | , , , , , , |
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| Format: | Artigo |
| Langue: | Inglês |
| Publié: |
2007
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| Sujets: | |
| Accès en ligne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2864119/ https://ncbi.nlm.nih.gov/pubmed/17898087 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.07-8430com |
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