A pertussis toxin sensitive G-protein-independent pathway is involved in serum amyloid A-induced formyl peptide receptor 2-mediated CCL2 production

Serum amyloid A (SAA) induced CCL2 production via a pertussis toxin (PTX)-insensitive pathway in human umbilical vein endothelial cells (HUVECs). SAA induced the activation of three MAPKs (ERK, p38 MAPK, and JNK), which were completely inhibited by knock-down of formyl peptide receptor 2 (FPR2). Inh...

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Bibliografiske detaljer
Main Authors: Lee, Ha Young, Kim, Sang Doo, Shim, Jae Woong, Kim, Hak Jung, Yun, Jeanho, Baek, Suk-Hwan, Kim, Koanhoi, Bae, Yoe-Sik
Format: Artigo
Sprog:Inglês
Udgivet: Korean Society for Biochemistry and Molecular Biology 2010
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Original Article
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2859329/
https://ncbi.nlm.nih.gov/pubmed/20177146
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3858/emm.2010.42.4.029
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