Candida albicans β-Glucan Exposure Is Controlled by the Fungal CEK1-Mediated Mitogen-Activated Protein Kinase Pathway That Modulates Immune Responses Triggered through Dectin-1

Innate immunity to Candida albicans depends upon the recognition of molecular patterns on the fungal cell wall. However, the masking of major components such as β-glucan seems to be a mechanism that fungi have evolved to avoid immune cell recognition through the dectin-1 receptor. Although the role...

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מידע ביבליוגרפי
Main Authors: Galán-Díez, Marta, Arana, David M., Serrano-Gómez, Diego, Kremer, Leonor, Casasnovas, José M., Ortega, Mara, Cuesta-Domínguez, Álvaro, Corbí, Angel L., Pla, Jesús, Fernández-Ruiz, Elena
פורמט: Artigo
שפה:Inglês
יצא לאור: American Society for Microbiology (ASM) 2010
נושאים:
Fungal and Parasitic Infections
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC2849429/
https://ncbi.nlm.nih.gov/pubmed/20100861
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/IAI.00989-09
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