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Stress-sensitive Regulation of IFRD1 mRNA Decay Is Mediated by an Upstream Open Reading Frame

In this report, we demonstrate that cellular stress regulates expression of IFRD1 by a post-transcriptional control mechanism. IFRD1 mRNA and protein are elevated in tunicamycin-treated human kidney epithelial cells via stabilization of the mRNA. IFRD1 mRNA instability in resting cells requires tran...

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Bibliografiske detaljer
Main Authors: Zhao, Chenyang, Datta, Shyamasree, Mandal, Palash, Xu, Shuqing, Hamilton, Thomas
Format: Artigo
Sprog:Inglês
Udgivet: American Society for Biochemistry and Molecular Biology 2010
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2838277/
https://ncbi.nlm.nih.gov/pubmed/20080976
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M109.070920
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