Long-term exposure of rheumatoid arthritis synovial fibroblasts to tumor necrosis factor alpha inhibits FasL-mediated apoptosis through activation of NF-κB and upregulation of the small ubiquitin-like modifier SUMO-1

Míreanna Comhchosúla

• Increased susceptibility of osteoarthritis tenocytes to FasL-induced apoptosis is associated with elevated expression of Fas receptor but no alterations in the expression of Sentrin-1/SUMO-1
  le: Machner, A, et al.
  Foilsithe: (2003)
• Mechanisms of resistance against Fas-induced apoptosis in rheumatoid arthritis synovial fibroblasts
  le: Pap, T, et al.
  Foilsithe: (2003)
• Modification of nuclear PML protein by SUMO-1 regulates Fas-induced apoptosis in rheumatoid arthritis synovial fibroblasts
  le: Meinecke, Ingmar, et al.
  Foilsithe: (2007)
• Involvement of SUMO-1 in the regulation of apoptosis in prosthesis loosening fibroblasts
  le: Meinecke, I, et al.
  Foilsithe: (2004)
• Regulating apoptosis in fibroblasts
  le: Pap, T, et al.
  Foilsithe: (2005)