Transcriptional activation of homologous viral long terminal repeats by the human immunodeficiency virus type 1 or the human T-cell leukemia virus type I tat proteins occurs in the absence of de novo protein synthesis.

The genomes of human retroviruses [human immunodeficiency virus type 1 (HIV-1) and human T-cell leukemia virus (HTLV-I)] encode positive trans-activator proteins, named tat. In the presence of tat, the transcriptional activity of the homologous HIV-1 or HTLV-I long terminal repeat (LTR) promoter is...

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Publicado en:Proc Natl Acad Sci U S A
Autores principales: Jeang, K T, Shank, P R, Kumar, A
Formato: Artigo
Lenguaje:Inglês
Publicado: National Academy of Sciences 1988
Materias:
Research Article
Acceso en línea:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC282415/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2847157/
https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.85.21.8291
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