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Genome-wide end-sequenced BAC resources for the NOD/MrkTac(☆) and NOD/ShiLtJ(☆☆) mouse genomes
Non-obese diabetic (NOD) mice spontaneously develop type 1 diabetes (T1D) due to the progressive loss of insulin-secreting β-cells by an autoimmune driven process. NOD mice represent a valuable tool for studying the genetics of T1D and for evaluating therapeutic interventions. Here we describe the d...
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| Asıl Yazarlar: | , , , , , , , , , , , , , , , , , , , , , |
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| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
Academic Press
2010
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2824108/ https://ncbi.nlm.nih.gov/pubmed/19909804 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ygeno.2009.10.004 |
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