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Atypical protein kinase C activity is required for extracellular matrix degradation and invasion by Src-transformed cells
Atypical protein kinase C (aPKC) isoforms have been shown to mediate Src-dependent signaling in response to growth factor stimulation. To determine if aPKC activity contributes to the transformed phenotype of cells expressing oncogenic Src, we have examined the activity and function of aPKCs in 3T3...
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Главные авторы: | , , |
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Формат: | Artigo |
Язык: | Inglês |
Опубликовано: |
2009
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Предметы: | |
Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2782718/ https://ncbi.nlm.nih.gov/pubmed/19492416 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/jcp.21841 |
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