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Atypical protein kinase C activity is required for extracellular matrix degradation and invasion by Src-transformed cells

Atypical protein kinase C (aPKC) isoforms have been shown to mediate Src-dependent signaling in response to growth factor stimulation. To determine if aPKC activity contributes to the transformed phenotype of cells expressing oncogenic Src, we have examined the activity and function of aPKCs in 3T3...

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Bibliografski detalji
Glavni autori: Rodriguez, E., Dunham, E., Martin, G. S.
Format: Artigo
Jezik:Inglês
Izdano: 2009
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC2782718/
https://ncbi.nlm.nih.gov/pubmed/19492416
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/jcp.21841
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