Intra-cornu ammonis 1 administration of the human immunodeficiency virus-1 transcription factor Tat exacerbates the ethanol withdrawal syndrome in rodents and activates N-methyl-d-aspartate glutamate receptors to produce persisting spatial learning deficits

Human immunodeficiency virus-1 (HIV-1) infection may produce neurological deficits, such as cognitive decline, that may be worsened by concurrent ethanol (EtOH) abuse. Among the many biochemical cascades likely mediating HIV-1 associated neuronal injury is enhancement of N-methyl-d-aspartate (NMDA)...

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Hauptverfasser: Self, Rachel L., Smith, Katherine J., Butler, Tracy R., Pauly, James R., Prendergast, Mark A.
Format: Artigo
Sprache:Inglês
Veröffentlicht: 2009
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Article
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2773563/
https://ncbi.nlm.nih.gov/pubmed/19619615
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuroscience.2009.07.025
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