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X11β rescues memory and long-term potentiation deficits in Alzheimer's disease APPswe Tg2576 mice
Increased production and deposition of amyloid β-protein (Aβ) are believed to be key pathogenic events in Alzheimer's disease. As such, routes for lowering cerebral Aβ levels represent potential therapeutic targets for Alzheimer's disease. X11β is a neuronal adaptor protein that binds to t...
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| Main Authors: | , , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Oxford University Press
2009
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2773264/ https://ncbi.nlm.nih.gov/pubmed/19744962 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/hmg/ddp408 |
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