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Toll-like receptor 2 (TLR2) deficiency leads to increased Th17 infiltrates in experimental brain abscesses
TLR2 plays a pivotal role in recognizing S. aureus, a common etiologic agent of CNS parenchymal infections, such as brain abscess. We previously reported that brain abscesses of TLR2 knockout (KO) mice exhibited elevated IL-17 levels, suggesting the presence of an alternative pathway available to re...
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| Hlavní autoři: | , , , , , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
2009
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2713313/ https://ncbi.nlm.nih.gov/pubmed/19454709 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.0802656 |
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