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Inhibition of TRPC1/TRPC3 by PKG contributes to NO-mediated vasorelaxation
Nitric oxide (NO) inhibits transient receptor potential channel 3 (TRPC3) channels via a PKG-dependent mechanism. We sought to determine 1) whether NO inhibition of TRPC3 occurs in freshly isolated smooth muscle cells (SMC); and 2) whether NO inhibition of TRPC3 channels contributes to NO-mediated v...
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| Autors principals: | , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Physiological Society
2009
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2711742/ https://ncbi.nlm.nih.gov/pubmed/19502552 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.01130.2008 |
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