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Induction of the Tat-binding protein 1 gene accompanies the disabling of oncogenic erbB receptor tyrosine kinases

Conversion of a malignant phenotype into a more normal one can be accomplished either by down-regulation of erbB family surface receptors or by creating inactive erbB heterodimers on the cell surface. In this report, we report the identification and cloning of differentially expressed genes from ant...

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Detalhes bibliográficos
Main Authors: Park, Byeong-Woo, O’Rourke, Donald M., Wang, Qiang, Davis, James G., Post, Andrew, Qian, Xiaolan, Greene, Mark I.
Formato: Artigo
Idioma:Inglês
Publicado em: The National Academy of Sciences 1999
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC26899/
https://ncbi.nlm.nih.gov/pubmed/10339605
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