טוען...
Gfi1 integrates progenitor versus granulocytic transcriptional programming
In patients with severe congenital neutropenia (SCN) and mice with growth factor independent-1 (Gfi1) loss of function, arrested myeloid progenitors accumulate, whereas terminal granulopoiesis is blocked. One might assume that Gfi-null progenitors accumulate because they lack the ability to differen...
שמור ב:
| Main Authors: | , , , , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Society of Hematology
2009
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2689047/ https://ncbi.nlm.nih.gov/pubmed/19346496 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2008-09-179747 |
| תגים: |
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