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Ischemic Preconditioning Targets the Respiration of Synaptic Mitochondria via Protein Kinase Cε

In the brain, ischemic preconditioning (IPC) diminishes mitochondrial dysfunction after ischemia and confers neuroprotection. Activation of ε protein kinase C (εPKC) has been proposed to be a key neuroprotective pathway during IPC. We tested the hypothesis that IPC increases the levels of εPKC in sy...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Dave, Kunjan R., DeFazio, R. Anthony, Raval, Ami P., Torraco, Alessandra, Saul, Isabel, Barrientos, Antoni, Perez-Pinzon, Miguel A.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Society for Neuroscience 2008
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2678917/
https://ncbi.nlm.nih.gov/pubmed/18417696
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.5471-07.2008
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