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Glial–Cytokine–Neuronal Interactions Underlying the Mechanisms of Persistent Pain
The emerging literature implicates a role for glia/cytokines in persistent pain. However, the mechanisms by which these non-neural elements contribute to CNS activity-dependent plasticity and pain are unclear. Using a trigeminal model of inflammatory hyperalgesia, here we provide evidence that demon...
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| Autors principals: | , , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Society for Neuroscience
2007
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2676443/ https://ncbi.nlm.nih.gov/pubmed/17537972 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.0176-07.2007 |
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