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Neurotensin in the Ventral Pallidum Increases Extracellular γ-Aminobutyric Acid and Differentially Affects Cue- and Cocaine-Primed Reinstatement

Cocaine-primed reinstatement is an animal model of drug relapse. The neurocircuitry underlying cocaine-primed reinstatement includes a decrease in GABA in the ventral pallidum (VP) that is inhibited by a μ opioid receptor antagonist, suggesting that opioid peptides colocalized with GABA in the proje...

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Detalles Bibliográficos
Main Authors: Torregrossa, Mary M., Kalivas, Peter W.
Formato: Artigo
Idioma:Inglês
Publicado: 2008
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC2672956/
https://ncbi.nlm.nih.gov/pubmed/18252810
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1124/jpet.107.130310
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