Premature senescence of highly proliferative endothelial progenitor cells is induced by tumor necrosis factor-α via the p38 mitogen-activated protein kinase pathway

Senescence of endothelial cells increases with systemic aging and is thought to contribute to the development of atherosclerosis. Cell therapy with highly proliferative endothelial progenitor cells (EPCs) is an emerging therapeutic option to promote endothelial regeneration, but little is known abou...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Zhang, Yanmin, Herbert, Brittney-Shea, Rajashekhar, Gangaraju, Ingram, David A., Yoder, Mervin C., Clauss, Matthias, Rehman, Jalees
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: The Federation of American Societies for Experimental Biology 2009
Aiheet:
Research Communications
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2669419/
https://ncbi.nlm.nih.gov/pubmed/19124561
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.08-110296
Tagit: Lisää tagi
Ei tageja, Lisää ensimmäinen tagi!

Samankaltaisia teoksia