Premature senescence of highly proliferative endothelial progenitor cells is induced by tumor necrosis factor-α via the p38 mitogen-activated protein kinase pathway

Senescence of endothelial cells increases with systemic aging and is thought to contribute to the development of atherosclerosis. Cell therapy with highly proliferative endothelial progenitor cells (EPCs) is an emerging therapeutic option to promote endothelial regeneration, but little is known abou...

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Detalhes bibliográficos
Main Authors: Zhang, Yanmin, Herbert, Brittney-Shea, Rajashekhar, Gangaraju, Ingram, David A., Yoder, Mervin C., Clauss, Matthias, Rehman, Jalees
Formato: Artigo
Idioma:Inglês
Publicado em: The Federation of American Societies for Experimental Biology 2009
Assuntos:
Research Communications
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2669419/
https://ncbi.nlm.nih.gov/pubmed/19124561
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.08-110296
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