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Regulation of the ClC-2 Lung Epithelial Chloride Channel by Glycosylation of SP1
Chloride channel–2 (ClC-2) is a pH- and voltage-activated chloride channel that is highly expressed in mammalian fetal airway epithelia during the period of maximal fluid secretion. A high level of luminal ClC-2 protein expression is maintained by the SP1 transcription factor until SP1 and ClC-2 dec...
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| Hauptverfasser: | , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
American Thoracic Society
2006
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2644236/ https://ncbi.nlm.nih.gov/pubmed/16456185 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1165/rcmb.2005-0442OC |
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