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Uremia induces proximal tubular cytoresistance and heme oxygenase-1 expression in the absence of acute kidney injury
Acute kidney injury (AKI) induces adaptive responses within proximal tubular (PT) cells that serve to protect them from further ischemic or toxic damage. However, it is not known whether uremia, a potential consequence of AKI, independently alters susceptibility to tubular injury. To address this is...
Tallennettuna:
| Päätekijä: | |
|---|---|
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
American Physiological Society
2009
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2643867/ https://ncbi.nlm.nih.gov/pubmed/19036845 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.90645.2008 |
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