Cell surface expression of the epithelial Na channel and a mutant causing Liddle syndrome: A quantitative approach
The epithelial amiloride-sensitive sodium channel (ENaC) controls transepithelial Na(+) movement in Na(+)-transporting epithelia and is associated with Liddle syndrome, an autosomal dominant form of salt-sensitive hypertension. Detailed analysis of ENaC channel properties and the functional conseque...
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| Vydáno v: | Proc Natl Acad Sci U S A |
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| Hlavní autoři: | , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
National Academy of Sciences
1996
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC26411/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8986818/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.93.26.15370 |
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