ロード中...
Mouse models for preeclampsia: disruption of redox-regulated signaling
The concept that oxidative stress contributes to the development of human preeclampsia has never been tested in genetically-defined animal models. Homozygous deletion of catechol-O-methyl transferase (Comt-/-) in pregnant mice leads to human preeclampsia-like symptoms (high blood pressure, albuminur...
保存先:
| 主要な著者: | , , |
|---|---|
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
BioMed Central
2009
|
| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2632643/ https://ncbi.nlm.nih.gov/pubmed/19146669 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1477-7827-7-4 |
| タグ: |
タグ追加
タグなし, このレコードへの初めてのタグを付けませんか!
|