The carboxyl-terminal region of staphylococcal enterotoxin type A is required for a fully active molecule.
Staphylococcal enterotoxin type A (SEA) gene (sea+) mutations were constructed by exonuclease III digestion or cassette mutagenesis. Five different sea mutations that had 1, 3, 7, 39, and 65 codons deleted from the 3' end of sea+ were identified and confirmed by restriction enzyme and nucleotide seq...
保存先:
| 出版年: | Infect Immun |
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| 主要な著者: | , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Microbiology (ASM)
1991
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC257976/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/1903773/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/iai.59.6.2126-2134.1991 |
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