Mutations in the Vaccinia Virus A33R and B5R Envelope Proteins That Enhance Release of Extracellular Virions and Eliminate Formation of Actin-Containing Microvilli without Preventing Tyrosine Phosphorylation of the A36R Protein

The spread of vaccinia virus in cell cultures is mediated by virions that adhere to the tips of specialized actin-containing microvilli and also by virions that are released into the medium. The use of a small plaque-forming A36R gene deletion mutant to select spontaneous second-site mutants exhibit...

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Bibliografische Detailangaben

Veröffentlicht in:	J Virol
Hauptverfasser:	Katz, Ehud, Ward, Brian M., Weisberg, Andrea S., Moss, Bernard
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	American Society for Microbiology (ASM) 2003
Schlagworte:	Virus-Cell Interactions
Online-Zugang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC254252/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/14581563/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/JVI.77.22.12266-12275.2003
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