Trigeminal ganglion infection by thymidine kinase-negative mutants of herpes simplex virus after in vivo complementation.

Lignende værker

• Latency-associated transcript but not reactivatable virus is present in sensory ganglion neurons after inoculation of thymidine kinase-negative mutants of herpes simplex virus type 1.
  af: Tenser, R B, et al.
  Udgivet: (1989)
• Thymidine kinase-negative herpes simplex virus mutants establish latency in mouse trigeminal ganglia but do not reactivate.
  af: Coen, D M, et al.
  Udgivet: (1989)
• Hepatic infection by thymidine kinase-positive and thymidine kinase-negative herpes simplex virus after partial hepatectomy.
  af: Fang, Z Y, et al.
  Udgivet: (1983)
• Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleoside.
  af: Tenser, R B, et al.
  Udgivet: (1996)
• The pathogenicity of thymidine kinase-deficient mutants of herpes simplex virus in mice.
  af: Field, H. J., et al.
  Udgivet: (1978)