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Silencing the Kir4.1 Potassium Channel Subunit in Satellite Glial Cells of the Rat Trigeminal Ganglion Results in Pain-Like Behavior in the Absence of Nerve Injury

Growing evidence suggests that changes in the ion buffering capacity of glial cells can give rise to neuropathic pain. In the CNS, potassium ion (K(+)) buffering is dependent on the glia-specific inward rectifying K(+) channel Kir4.1. We recently reported that the satellite glial cells that surround...

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Sonraí Bibleagrafaíochta
Main Authors: Vit, Jean-Philippe, Ohara, Peter T., Bhargava, Aditi, Kelley, Kanwar, Jasmin, Luc
Formáid: Artigo
Teanga:Inglês
Foilsithe: Society for Neuroscience 2008
Ábhair:
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC2533133/
https://ncbi.nlm.nih.gov/pubmed/18417695
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.5053-07.2008
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