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Silencing the Kir4.1 Potassium Channel Subunit in Satellite Glial Cells of the Rat Trigeminal Ganglion Results in Pain-Like Behavior in the Absence of Nerve Injury

Growing evidence suggests that changes in the ion buffering capacity of glial cells can give rise to neuropathic pain. In the CNS, potassium ion (K(+)) buffering is dependent on the glia-specific inward rectifying K(+) channel Kir4.1. We recently reported that the satellite glial cells that surround...

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Bibliografske podrobnosti
Main Authors: Vit, Jean-Philippe, Ohara, Peter T., Bhargava, Aditi, Kelley, Kanwar, Jasmin, Luc
Format: Artigo
Jezik:Inglês
Izdano: Society for Neuroscience 2008
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC2533133/
https://ncbi.nlm.nih.gov/pubmed/18417695
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.5053-07.2008
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