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Dysregulation of microRNAs after myocardial infarction reveals a role of miR-29 in cardiac fibrosis

Acute myocardial infarction (MI) due to coronary artery occlusion is accompanied by a pathological remodeling response that includes hypertrophic cardiac growth and fibrosis, which impair cardiac contractility. Previously, we showed that cardiac hypertrophy and heart failure are accompanied by chara...

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Detalhes bibliográficos
Main Authors: van Rooij, Eva, Sutherland, Lillian B., Thatcher, Jeffrey E., DiMaio, J. Michael, Naseem, R. Haris, Marshall, William S., Hill, Joseph A., Olson, Eric N.
Formato: Artigo
Idioma:Inglês
Publicado em: National Academy of Sciences 2008
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2529064/
https://ncbi.nlm.nih.gov/pubmed/18723672
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0805038105
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