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Receptor trafficking controls weak signal delivery: a strategy used by c-Met for STAT3 nuclear accumulation
C-Met, the receptor of hepatocyte growth factor (HGF), through overexpression or mutation, is a major protooncogene that provides an attractive molecular target for cancer therapy. HGF/c-Met–induced tumorigenesis is dependent, in part, on the transcription factor and oncogene signal transducer and a...
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| Main Authors: | , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
The Rockefeller University Press
2008
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| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2528569/ https://ncbi.nlm.nih.gov/pubmed/18779368 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1083/jcb.200806076 |
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