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Tumor Cell Dormancy Induced by p38(SAPK) and ER-Stress Signaling: An Adaptive Advantage for Metastatic Cells?
The mechanisms that determine whether a tumor cell that has disseminated to a secondary site will resume growth immediately, die or enter a state of dormancy are poorly understood. Although tumor dormancy represents a common clinical finding, studying the mechanisms behind this stage of tumor progre...
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| Autors principals: | , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2006
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2516910/ https://ncbi.nlm.nih.gov/pubmed/16861922 |
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