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Recombination between feline leukemia virus subgroup B or C and endogenous env elements alters the in vitro biological activities of the viruses.

An important question in feline leukemia virus (FeLV) pathogenesis is whether, as in murine leukemia virus infection, homologous recombination between the infecting FeLV and the noninfectious endogenous FeLV-like proviruses serves as a significant base for the generation of proximal pathogens. To be...

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Main Authors: Pandey, R, Ghosh, A K, Kumar, D V, Bachman, B A, Shibata, D, Roy-Burman, P
格式: Artigo
語言:Inglês
出版: 1991
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在線閱讀:https://ncbi.nlm.nih.gov/pmc/articles/PMC250696/
https://ncbi.nlm.nih.gov/pubmed/1658356
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