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Mitochondrial Ca(2+) Overload Underlies Aβ Oligomers Neurotoxicity Providing an Unexpected Mechanism of Neuroprotection by NSAIDs
Dysregulation of intracellular Ca(2+) homeostasis may underlie amyloid β peptide (Aβ) toxicity in Alzheimer's Disease (AD) but the mechanism is unknown. In search for this mechanism we found that Aβ(1–42) oligomers, the assembly state correlating best with cognitive decline in AD, but not Aβ fi...
שמור ב:
| Main Authors: | , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Public Library of Science
2008
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2447871/ https://ncbi.nlm.nih.gov/pubmed/18648507 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0002718 |
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