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Systemic Inflammation Exacerbates Behavioral and Histopathological Consequences of Isolated Traumatic Brain Injury In Rats

The proinflammatory cytokine interleukin-1β (IL-1β) is induced rapidly after traumatic brain injury (TBI) and contributes to the inflammatory events that lead to neuronal loss. Although an important source of IL-1β is from the injured brain itself, in patients with multiple organ trauma (polytrauma)...

תיאור מלא

שמור ב:
מידע ביבליוגרפי
Main Authors: Utagawa, Akira, Truettner, Jessie S., Dietrich, W. Dalton, Bramlett, Helen M.
פורמט: Artigo
שפה:Inglês
יצא לאור: 2008
נושאים:
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC2435258/
https://ncbi.nlm.nih.gov/pubmed/18355811
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.expneurol.2008.02.001
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