Elevated free nitrotyrosine levels, but not protein-bound nitrotyrosine or hydroxyl radicals, throughout amyotrophic lateral sclerosis (ALS)-like disease implicate tyrosine nitration as an aberrant in vivo property of one familial ALS-linked superoxide dismutase 1 mutant

समान संसाधन

• Metal Deficiency Increases Aberrant Hydrophobicity of Mutant Superoxide Dismutases That Cause Amyotrophic Lateral Sclerosis
  द्वारा: Tiwari, Ashutosh, और अन्य
  प्रकाशित: (2009)
• Role of Copper,Zinc-Superoxide Dismutase in Catalyzing Nitrotyrosine Formation in Murine Liver
  द्वारा: Zhu, Jian-Hong, और अन्य
  प्रकाशित: (2008)
• Role of superoxide dismutase in in vivo and in vitro nitrate tolerance
  द्वारा: Münzel, Thomas, और अन्य
  प्रकाशित: (1999)
• Mutant Superoxide Dismutase 1 Forms Aggregates in the Brain Mitochondrial Matrix of Amyotrophic Lateral Sclerosis Mice
  द्वारा: Vijayvergiya, Chetan, और अन्य
  प्रकाशित: (2005)
• Superoxide dismutase 1 with mutations linked to familial amyotrophic lateral sclerosis possesses significant activity.
  द्वारा: Borchelt, D R, और अन्य
  प्रकाशित: (1994)