Extracellular SOD protects the heart against oxidative stress and hypertrophy after myocardial infarction

Extracellular SOD (EC-SOD) contributes only a small fraction to total SOD activity in the heart, but is strategically located to scavenge free radicals in the extracellular compartment. EC-SOD expression is decreased in myocardial infarction (MI)-induced failing heart, but whether EC-SOD can abrogat...

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Detaylı Bibliyografya
Asıl Yazarlar: van Deel, Elza D., Lu, Zhongbing, Xu, Xin, Zhu, Guangshuo, Hu, Xinli, Oury, Tim D., Bache, Robert J, Duncker, Dirk J., Chen, Yingjie
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2007
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2386859/
https://ncbi.nlm.nih.gov/pubmed/18206658
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.freeradbiomed.2007.12.007
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