Escape from in vivo restriction of Moloney mink cell focus-inducing viruses driven by the Mo+PyF101 long terminal repeat (LTR) by LTR alterations.

Benzer Materyaller

• Recombinant mink cell focus-inducing virus and long terminal repeat alterations accompany the increased leukemogenicity of the Mo+PyF101 variant of Moloney murine leukemia virus after intraperitoneal inoculation.
  Yazar:: Belli, B, ve diğerleri
  Baskı/Yayın Bilgisi: (1995)
• Appearance of Mink Cell Focus-Inducing Recombinants during In Vivo Infection by Moloney Murine Leukemia Virus (M-MuLV) or the Mo+PyF101 M-MuLV Enhancer Variant: Implications for Sites of Generation and Roles in Leukemogenesis
  Yazar:: Lander, Jeffrey K., ve diğerleri
  Baskı/Yayın Bilgisi: (1999)
• Sequences responsible for erythroid and lymphoid leukemia in the long terminal repeats of Friend-mink cell focus-forming and Moloney murine leukemia viruses.
  Yazar:: Ishimoto, A, ve diğerleri
  Baskı/Yayın Bilgisi: (1987)
• Envelope gene and long terminal repeat determine the different biological properties of Rauscher, Friend, and Moloney mink cell focus-inducing viruses.
  Yazar:: Vogt, M, ve diğerleri
  Baskı/Yayın Bilgisi: (1985)
• Negative regulation of the 5' long terminal repeat (LTR) by the 3' LTR in the murine proviral genome.
  Yazar:: Gama Sosa, M A, ve diğerleri
  Baskı/Yayın Bilgisi: (1994)