Chronic human immunodeficiency virus type 1 infection stimulates distinct NF-kappa B/rel DNA binding activities in myelomonoblastic cells.

Podobne zapisy

• Regulation of human immunodeficiency virus type 1 and cytokine gene expression in myeloid cells by NF-kappa B/Rel transcription factors.
  od: Roulston, A, i wsp.
  Wydane: (1995)
• Chronic human immunodeficiency virus type 1 infection of myeloid cells disrupts the autoregulatory control of the NF-kappaB/Rel pathway via enhanced IkappaBalpha degradation.
  od: DeLuca, C, i wsp.
  Wydane: (1996)
• Interaction of the v-rel protein with an NF-kappa B DNA binding site.
  od: Kabrun, N, i wsp.
  Wydane: (1991)
• I kappa B alpha-mediated inhibition of v-Rel DNA binding requires direct interaction with the RXXRXRXXC Rel/kappa B DNA-binding motif.
  od: Kumar, S, i wsp.
  Wydane: (1993)
• Selection of optimal kappa B/Rel DNA-binding motifs: interaction of both subunits of NF-kappa B with DNA is required for transcriptional activation.
  od: Kunsch, C, i wsp.
  Wydane: (1992)