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Use-dependent control of presynaptic calcium signalling at central synapses
Voltage-gated Ca(2+) channels activated by action potentials evoke Ca(2+) entry into presynaptic terminals thus briefly distorting the resting Ca(2+) concentration. When this happens, a number of processes are initiated to re-establish the Ca(2+) equilibrium. During the post-spike period, the increa...
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| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
Blackwell Science Inc
2007
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| Online dostop: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2375749/ https://ncbi.nlm.nih.gov/pubmed/17523936 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1469-7580.2007.00728.x |
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