Erythropoietin receptor (EpoR)-dependent mitogenicity of spleen focus-forming virus correlates with viral pathogenicity and processing of env protein but not with formation of gp52-EpoR complexes in the endoplasmic reticulum.

Recent evidence suggests that interactions between spleen focus-forming virus (SFFV) env products and the erythropoietin receptor (EpoR) are responsible for viral pathogenicity. Infection of factor-dependent cell lines expressing epoR (the cloned gene for EpoR) with SFFVP is mitogenic, generating ce...

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Veröffentlicht in:	J Virol
Hauptverfasser:	Wang, Y, Kayman, S C, Li, J P, Pinter, A
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	American Society for Microbiology (ASM) 1993
Schlagworte:	Research Article
Online-Zugang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC237500/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8437218/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.67.3.1322-1327.1993
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