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Nitric oxide, leukocytes and microvascular permeability: causality or bystanders?
Increased microvascular permeability resulting in tissue edema is a hallmark of sepsis-related microcirculatory failure, and leukocyte–endothelium interaction is thought to assume major importance in this context. However, the role of nitric oxide (NO) in the interplay of inflammation, leukocyte–end...
Gorde:
| Egile Nagusiak: | , , |
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| Formatua: | Artigo |
| Hizkuntza: | Inglês |
| Argitaratua: |
BioMed Central
2008
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| Gaiak: | |
| Sarrera elektronikoa: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2374610/ https://ncbi.nlm.nih.gov/pubmed/18226179 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/cc6214 |
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