Upregulation of bcl-2 by the Epstein-Barr virus latent membrane protein LMP1: a B-cell-specific response that is delayed relative to NF-kappa B activation and to induction of cell surface markers.

An ability of the Epstein-Barr virus latent membrane protein LMP1 to enhance the survival of infected B cells through upregulation of the bcl-2 oncogene was first suggested by experiments involving gene transfection and the selection of stable LMP1+ clones (S. Henderson, M. Rowe, C. Gregory, F. Wang...

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Detaylı Bibliyografya

Yayımlandı:	J Virol
Asıl Yazarlar:	Rowe, M, Peng-Pilon, M, Huen, D S, Hardy, R, Croom-Carter, D, Lundgren, E, Rickinson, A B
Materyal Türü:	Artigo
Dil:	Inglês
Baskı/Yayın Bilgisi:	American Society for Microbiology (ASM) 1994
Konular:	Research Article
Online Erişim:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC236961/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7520093/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.68.9.5602-5612.1994
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