The tat/rev intron of human immunodeficiency virus type 1 is inefficiently spliced because of suboptimal signals in the 3' splice site.

Ítems similars

• Identification of positive and negative splicing regulatory elements within the terminal tat-rev exon of human immunodeficiency virus type 1.
  per: Staffa, A, et al.
  Publicat: (1995)
• Control of Human Immunodeficiency Virus Type 1 RNA Metabolism: Role of Splice Sites and Intron Sequences in Unspliced Viral RNA Subcellular Distribution
  per: Séguin, Béatrice, et al.
  Publicat: (1998)
• Splicing efficiency of human immunodeficiency virus type 1 tat RNA is determined by both a suboptimal 3' splice site and a 10 nucleotide exon splicing silencer element located within tat exon 2.
  per: Si, Z, et al.
  Publicat: (1997)
• Splicing of constitutive upstream introns is essential for the recognition of intra-exonic suboptimal splice sites in the thrombopoietin gene
  per: Romano, Maurizio, et al.
  Publicat: (2001)
• Tat and Rev: positive modulators of human immunodeficiency virus gene expression
  per: Rosen, Craig A.
  Publicat: (2018)