Deletion of an amino-terminal sequence beta-catenin in vivo and promotes hyperphosporylation of the adenomatous polyposis coli tumor suppressor protein.

Regulation of cell adhesion and cell signaling by beta-catenin occurs through a mechanism likely involving the targeted degradation of the protein. Deletional analysis was used to generate a beta-catenin refractory to rapid turnover and to examine its effects on complexes containing either cadherin...

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Veröffentlicht in:	Mol Cell Biol
Hauptverfasser:	Munemitsu, S, Albert, I, Rubinfeld, B, Polakis, P
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	Taylor & Francis 1996
Schlagworte:	Research Article
Online-Zugang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC231405/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8754807/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.16.8.4088
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